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1Nutrition and Endocrine Research Center and Obesity Research Center, Research Institute for Endocrine Sciences, Tehran, Iran.
2Department of Clinical Nutrition and Diet Therapy, Faculty of Nutrition Sciences and Food Technology, National Nutrition and Food Technology Research Institute, Tehran, Iran.
3Research Center for Gastroenterology and Liver Disease, Department of Gastroenterology and Liver Diseases, Tehran, Iran.
4Endocrine Research Center, Research Institute for Endocrine Sciences, Shahid Beheshti University of Medical Sciences, Tehran, Iran.
Copyright © 2015 Korean Endocrine Society
This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
CONFLICTS OF INTEREST: No potential conflict of interest relevant to this article was reported.
Values are expressed as age-adjusted mean±SEM unless otherwise indicated.
HpSAg, Helicobacter pylori stool antigen; PGI, pepsinogen I; PGII, pepsinogen II; HOMA-IR, homeostasis model assessment of insulin resistance; HDL-C, high density lipoprotein cholesterol; LDL-C, low density lipoprotein cholesterol; hs-CRP, high-sensitive C-reactive protein.
aP values compared the mean values across quartile categories of PGI/PGII ratio using analysis of covariance (with adjustments for age, sex, antidiabetic and antilipidemic drugs, duration of diabetes) or Jonckheere-Terpstra test.
Pearson correlation test was used.
HpSAg, Helicobacter pylori stool antigen; PGI, pepsinogen I; PGII, pepsinogen II; HOMA-IR, homeostasis model assessment of insulin resistance; HDL-C, high density lipoprotein cholesterol; LDL-C, low density lipoprotein cholesterol; hs-CRP, high-sensitive C-reactive protein.