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Corrigendum
Corrigendum: Correction of Figure. High Cardiorespiratory Fitness Protects against Molecular Impairments of Metabolism, Heart, and Brain with Higher Efficacy in Obesity-Induced Premature Aging
Patcharapong Pantiya1,2,3*, Chanisa Thonusin1,2,3*, Natticha Sumneang1,2,3, Benjamin Ongnok1,2,3, Titikorn Chunchai1,3, Sasiwan Kerdphoo1,3, Thidarat Jaiwongkam1,3, Busarin Arunsak1,3, Natthaphat Siri-Angkul1,2,3, Sirawit Sriwichaiin1,2,3, Nipon Chattipakorn1,2,3, Siriporn C. Chattipakorn1,2,4

DOI: https://doi.org/10.3803/EnM.2025.401
Published online: June 23, 2025

1Neurophysiology Unit, Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

2Cardiac Electrophysiology Unit, Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

3Center of Excellence in Cardiac Electrophysiology Research, Chiang Mai University, Chiang Mai, Thailand

4Department of Oral Biology and Diagnostic Sciences, Faculty of Dentistry, Chiang Mai University, Chiang Mai, Thailand

Corresponding authors: Siriporn C. Chattipakorn. Neurophysiology Unit, Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, 110 Intawaroros Road, Si Phum, Muang, Chiang Mai 50200, Thailand Tel: +66-53-935329, Fax: +66-53-935368, E-mail: siriporn.c@cmu.ac.th
*These authors contributed equally to this work.

Copyright © 2025 Korean Endocrine Society

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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This corrects the article "High Cardiorespiratory Fitness Protects against Molecular Impairments of Metabolism, Heart, and Brain with Higher Efficacy in Obesity-Induced Premature Aging" on page 630.
Endocrinol Metab 2022;37:630-40
https://doi.org/10.3803/EnM.2022.1430
The authors would like to point out that the wrong version of Supplemental Fig. S2 has appeared in the published article. New representative Western blot image of the mitochondrial parkin in cardiac tissue has been replaced. The corrected figure is presented in the below corrigendum and this correction does not alter the conclusions drawn from this work. The authors sincerely apologize for this error.
Supplemental Fig. S2.
Representative picture of Western blotting in cardiac tissue of normal diet-fed rats (ND) versus high-fat diet-fed rats (HFD) at follow-up (week 28). CPT1, carnitine palmitoyl transferase I; PGC-1α, peroxisome proliferator-activated receptor gamma coactivator-1α; p-AMPK, phosphorylated-activated protein kinase; AMPK, activated protein kinase; p-IRS, phosphorylated-insulin receptor substrate 1; IRS, insulin receptor substrate 1; MFN1, mitofusin 1; MFN2, mitofusin 2; OPA1, optic atrophy 1; DRP1, dynamin-related protein 1; p-DRP1, phosphorylated-dynamin-related protein; PINK1, PTEN-induced kinase 1; LC3-II, light chain 3-II; Bax, Bcl-2-associated X protein; Bcl, B-cell lymphoma; GPX4, glutathione peroxidase 4; SOD2, superoxide dismutase 2; MDA, malondialdehyde; TNF-α, tumor necrosis factor-α; p-NFκB, phosphorylated-nuclear factor kappa-light-chain-enhancer of activated B cells; NFκB, nuclear factor kappa-light-chain-enhancer of activated B cells; sRAGE, soluble-receptor for advanced glycation end product; VDAC, voltage-dependent anion channel; GAPDH, glyceraldehyde 3-phosphate dehydrogenase.
enm-2025-401f1.jpg

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      Corrigendum: Correction of Figure. High Cardiorespiratory Fitness Protects against Molecular Impairments of Metabolism, Heart, and Brain with Higher Efficacy in Obesity-Induced Premature Aging
      Image
      Supplemental Fig. S2. Representative picture of Western blotting in cardiac tissue of normal diet-fed rats (ND) versus high-fat diet-fed rats (HFD) at follow-up (week 28). CPT1, carnitine palmitoyl transferase I; PGC-1α, peroxisome proliferator-activated receptor gamma coactivator-1α; p-AMPK, phosphorylated-activated protein kinase; AMPK, activated protein kinase; p-IRS, phosphorylated-insulin receptor substrate 1; IRS, insulin receptor substrate 1; MFN1, mitofusin 1; MFN2, mitofusin 2; OPA1, optic atrophy 1; DRP1, dynamin-related protein 1; p-DRP1, phosphorylated-dynamin-related protein; PINK1, PTEN-induced kinase 1; LC3-II, light chain 3-II; Bax, Bcl-2-associated X protein; Bcl, B-cell lymphoma; GPX4, glutathione peroxidase 4; SOD2, superoxide dismutase 2; MDA, malondialdehyde; TNF-α, tumor necrosis factor-α; p-NFκB, phosphorylated-nuclear factor kappa-light-chain-enhancer of activated B cells; NFκB, nuclear factor kappa-light-chain-enhancer of activated B cells; sRAGE, soluble-receptor for advanced glycation end product; VDAC, voltage-dependent anion channel; GAPDH, glyceraldehyde 3-phosphate dehydrogenase.

      Supplemental Fig. S2.

      Corrigendum: Correction of Figure. High Cardiorespiratory Fitness Protects against Molecular Impairments of Metabolism, Heart, and Brain with Higher Efficacy in Obesity-Induced Premature Aging
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