Corrigendum: Correction of Figure. High Cardiorespiratory Fitness Protects against Molecular Impairments of Metabolism, Heart, and Brain with Higher Efficacy in Obesity-Induced Premature Aging

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Endocrinol Metab. 2025;40(4):657-658

Publication date (electronic) : 2025 June 23

doi : https://doi.org/10.3803/EnM.2025.401

Patcharapong Pantiya ¹^,²^,³^,^*, Chanisa Thonusin ¹^,²^,³^,^*, Natticha Sumneang ¹^,²^,³, Benjamin Ongnok ¹^,²^,³, Titikorn Chunchai ¹^,³, Sasiwan Kerdphoo ¹^,³, Thidarat Jaiwongkam ¹^,³, Busarin Arunsak ¹^,³, Natthaphat Siri-Angkul ¹^,²^,³, Sirawit Sriwichaiin ¹^,²^,³, Nipon Chattipakorn ¹^,²^,³, Siriporn C. Chattipakorn^,¹^,²^,⁴

¹Neurophysiology Unit, Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

²Cardiac Electrophysiology Unit, Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai, Thailand

³Center of Excellence in Cardiac Electrophysiology Research, Chiang Mai University, Chiang Mai, Thailand

⁴Department of Oral Biology and Diagnostic Sciences, Faculty of Dentistry, Chiang Mai University, Chiang Mai, Thailand

Corresponding authors: Siriporn C. Chattipakorn. Neurophysiology Unit, Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, 110 Intawaroros Road, Si Phum, Muang, Chiang Mai 50200, Thailand Tel: +66-53-935329, Fax: +66-53-935368, E-mail: siriporn.c@cmu.ac.th

*These authors contributed equally to this work.

Endocrinol Metab 2022;37:630-40

https://doi.org/10.3803/EnM.2022.1430

The authors would like to point out that the wrong version of Supplemental Fig. S2 has appeared in the published article. New representative Western blot image of the mitochondrial parkin in cardiac tissue has been replaced. The corrected figure is presented in the below corrigendum and this correction does not alter the conclusions drawn from this work. The authors sincerely apologize for this error.

Supplemental Fig. S2.

Representative picture of Western blotting in cardiac tissue of normal diet-fed rats (ND) versus high-fat diet-fed rats (HFD) at follow-up (week 28). CPT1, carnitine palmitoyl transferase I; PGC-1α, peroxisome proliferator-activated receptor gamma coactivator-1α; p-AMPK, phosphorylated-activated protein kinase; AMPK, activated protein kinase; p-IRS, phosphorylated-insulin receptor substrate 1; IRS, insulin receptor substrate 1; MFN1, mitofusin 1; MFN2, mitofusin 2; OPA1, optic atrophy 1; DRP1, dynamin-related protein 1; p-DRP1, phosphorylated-dynamin-related protein; PINK1, PTEN-induced kinase 1; LC3-II, light chain 3-II; Bax, Bcl-2-associated X protein; Bcl, B-cell lymphoma; GPX4, glutathione peroxidase 4; SOD2, superoxide dismutase 2; MDA, malondialdehyde; TNF-α, tumor necrosis factor-α; p-NFκB, phosphorylated-nuclear factor kappa-light-chain-enhancer of activated B cells; NFκB, nuclear factor kappa-light-chain-enhancer of activated B cells; sRAGE, soluble-receptor for advanced glycation end product; VDAC, voltage-dependent anion channel; GAPDH, glyceraldehyde 3-phosphate dehydrogenase.

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